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This suggests that semaglutides cardioprotective mechanisms extend beyond weight reduction, potentially involving anti-inflammatory effects, improvements in endothelial function, blood pressure, and lipid modulation, or direct vascular and central nervous system pathways, although these mechanisms were not directly tested in this analysis

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Thus, the literature related to the chronology of ADRD pathophysiology implicates CBF as a critical early-stage target for attenuating neurodegenerative cascades prior to symptomatic changes in brain function

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