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Patients with vitiligo have been found to have not only significantly increased average levels of hydrogen peroxide (H 2 O 2 ) in their skin lesions, but also significantly lower measured activities of catalase, superoxide dismutase, glutathione peroxidase and glutathione S-transferase than in the control group, as well as significantly increased levels of malondialdehyde [9, 15, 16]

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A meta-analytic summary of systemic GSH indicates variable doseresponse and inconsistent biomarker effects, complicating mechanistic attribution