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This function is supported by findings that GGT deficiency, either clinical or experimental (GGT knockout mice), is associated with glutathionuria because of the absence of GGT activity on the apical surface of the cells in the proximal tubules of the kidneys and reduced content of cysteine in plasma and cells [13]

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292 The inhibition of apo A-I may be linked to atherosclerosis progression through chronic inflammation pathways