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Wang M, Kaufman RJ (2016) Protein misfolding in the endoplasmic reticulum as a conduit to human disease

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In this model, scheduled visits from weeks 4208 (that is, including the additional laboratory data collection visits for European patients) for the full analysis set were included as fixed effects, and interactions with treatment and baseline allowed the treatment effect and the importance of the baseline to vary over visits

sEVs derived from more invasive TNBC cell lines significantly increased the proliferation, migration, and invasion capacity of all three recipient cell lines (SKBR3, MDA-MB-231, and HCC1954)