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The establishment of this inflammatory tumor microenvironment (TME) is unique due to CNS immune privilege, which historically describes the brain as being shielded from systemic immune responses by the BBB (62)

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In our earlier studies, we found a significant decrease in GSH in T2D individuals, and among 12 different markers of OS measured, GSH impressively correlated with changes in HbA1c [27], suggesting that altering hyperglycemia rapidly results in changes in GSH

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