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Accumulating evidence has revealed that ferroptosis functions as a key factor in regulating Tfh cell activity in immunization mice models and controlling the preservation of viral-infected memory Tfh cells (98)

Although intensified glycemic control, blood pressure management, and inhibition of the reninangiotensin system (RAS) can slow DKD progression, its clinical course remains highly heterogeneous

Finally, early injection of tranexamic acid before admission and during hospitalization can reduce plasma syndecan-1 levels, alleviate endothelial glycocalyx damage, and enhance vascular repair mechanisms [131, 132]