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Biochemically, GGT is associated directly with GSH regulation and maintenance of its normal redox status in cells by breaking down extracellular GSH which cannot cross the cellular membrane to release cysteine and glycine building blocks which can more easily cross into the cell for de-novo synthesis of GSH during periods of oxidative stress [26]

Taken together, a clear distinction can be made between Clade 3 GS-like genes and those of Clades 1 and 2 based on their promoters, their spatial expression patterns, the presence of a signal peptide, and the likelihood of the sequence being upregulated in parasitic stages

Today 29 , 104087 (2024)

Rev Int Androl 22:2328