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In the model of intervertebral disc degeneration, allicin protected the nucleus pulposus cells against advanced oxidation protein productmodulated mitochondrial dysfunction and oxidative stress through the downregulation of the p38-MAPK signaling pathway [234]
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Studies in animal models have demonstrated BPC-157s effectiveness in accelerating the repair of tendon tears, making it a potential therapy for improving musculoskeletal health, particularly as we age
Both MK-801 and GLP-1 have been linked to improvements in glucose homeostasis in mice 20,21
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