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VPA poisoning is responsible for central nervous system manifestations ranging from ataxia, sedation and lethargy to coma, respiratory depression, seizures and intracranial hypertension [3]

however, changing to an alternative GLP-1 or GIP with a different excipient profile should be considered if hypersensitivity is unmanageable for the patient

In fact, for weeks, if not months, I kept wondering if the medication was really working

But research in recent years has shown that, it is also a precursor of the inhibitory neurotransmitter -aminobutyric acid (GABA), which can regulate the synthesis and secretion of glutamate and dopamine and plays an important role in the process of oxidative stress, apoptosis and neuroinflammation